middle cerebral artery infarction Search Results


90
Gallus BioPharmaceuticals internal carotid artery
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Johns Hopkins HealthCare middle cerebral artery occlusion
Middle Cerebral Artery Occlusion, supplied by Johns Hopkins HealthCare, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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middle cerebral artery occlusion - by Bioz Stars, 2026-08
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Nicolet Biomedical middle cerebral artery transcranial echo doppler eme companion
Middle Cerebral Artery Transcranial Echo Doppler Eme Companion, supplied by Nicolet Biomedical, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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BrainsGate Ltd occlusion of the middle cerebral artery (mcao)
Occlusion Of The Middle Cerebral Artery (Mcao), supplied by BrainsGate Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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occlusion of the middle cerebral artery (mcao) - by Bioz Stars, 2026-08
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PROACT Medical Ltd middle cerebral artery territory strokes
Subgroup analyses (functional outcome)
Middle Cerebral Artery Territory Strokes, supplied by PROACT Medical Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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middle cerebral artery territory strokes - by Bioz Stars, 2026-08
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Branston Ltd temporary middle cerebral artery occlusion
Subgroup analyses (functional outcome)
Temporary Middle Cerebral Artery Occlusion, supplied by Branston Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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temporary middle cerebral artery occlusion - by Bioz Stars, 2026-08
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Georg Thieme Verlag KG end-to-side anastomosis of superficial temporal artery to middle cerebral artery branch in the dog
Subgroup analyses (functional outcome)
End To Side Anastomosis Of Superficial Temporal Artery To Middle Cerebral Artery Branch In The Dog, supplied by Georg Thieme Verlag KG, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Anterio Inc middle cerebral artery (mca) injection
A 61-year old female presented with a right parietal hemorrhage, generalized tonic clonic seizure, and left hemiparesis. She was found to harbor an AVM at the site of hemorrhage and was treated with GKR 1 month after presentation. Several diagnostic cerebral angiogram images are presented. <t>A.)</t> <t>Anterio-posterior</t> (AP) projection of an internal carotid artery (ICA) injection. Poor detailed arterial, nidal, and venous resolution of the AVM are noted (White arrow). B.) Anterio-posterior (AP) projection of a selective middle cerebral artery <t>(MCA)</t> injection. Poor detailed arterial, nidal, and venous resolution of the AVM are noted (White arrow). C.) 3DRA right ICA injection with a right parietal AVM (white arrow) but with continued limited arterial, nidal, and venous anatomy. D.) Axial CBCT-A reconstruction with patient in the Leksell head frame. The anterior posts and posterior pins of the frame are seen. A right parietal AVM is visualized. E.) Magnified axial CBCT-A reconstruction. Excellent resolution of the complex arterial and venous structure of the AVM are noted (White arrow). F.) Screen shot image of a coronal CBCT-A reconstruction uploaded for planning on the Leksell stereotactic planning computer. The radiated field is demonstrated by the encircled areas at different radiation isodoses. Note that the draining vein is easily resolved and left out of the radiation field (White arrow). G.) 2-year follow-up AP projection of an ICA injection demonstrating complete obliteration of the previously seen AVM. H.) 2-year follow-up magnified axial CBCT-A reconstruction demonstrating complete obliteration of the previously seen AVM.
Middle Cerebral Artery (Mca) Injection, supplied by Anterio Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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middle cerebral artery (mca) injection - by Bioz Stars, 2026-08
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PETCO Animal Supplies Inc middle cerebral artery blood velocity (mca v mean)
End-tidal carbon dioxide tension and <t>middle</t> <t>cerebral</t> <t>artery</t> <t>blood</t> <t>velocity</t> (MCA Vmean) during normothermia, heat stress, and heat stress after end-tidal carbon dioxide (PETCO2) concentration was returned to pre-heat stress levels. The reduction in PETCO2 concentration during heat stress was completely abolished by the PETCO2 clamping procedure (panel a). Heat stress reduced MCA Vmean relative to normothermia. Restoration of PETCO2 to the normothermic level while subjects were heat stressed (heat stress + clamp) attenuated the decrease in MCA Vmean relative to control heat stress without the clamp; however MCA Vmean remained reduced when compared with normothermia (panel b). These data indicate that mechanisms other than reduced PETCO2 contribute to the reduced cerebral perfusion that occurs in heat-stressed individuals. *Significantly different relative to normothermia; §significantly different relative to control heat stress. Figure from Brothers et al. (2009b); republished with permission from Wiley-Blackwell.
Middle Cerebral Artery Blood Velocity (Mca V Mean), supplied by PETCO Animal Supplies Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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middle cerebral artery blood velocity (mca v mean) - by Bioz Stars, 2026-08
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Van Hoef Consulting middle cerebral artery occlusion models
End-tidal carbon dioxide tension and <t>middle</t> <t>cerebral</t> <t>artery</t> <t>blood</t> <t>velocity</t> (MCA Vmean) during normothermia, heat stress, and heat stress after end-tidal carbon dioxide (PETCO2) concentration was returned to pre-heat stress levels. The reduction in PETCO2 concentration during heat stress was completely abolished by the PETCO2 clamping procedure (panel a). Heat stress reduced MCA Vmean relative to normothermia. Restoration of PETCO2 to the normothermic level while subjects were heat stressed (heat stress + clamp) attenuated the decrease in MCA Vmean relative to control heat stress without the clamp; however MCA Vmean remained reduced when compared with normothermia (panel b). These data indicate that mechanisms other than reduced PETCO2 contribute to the reduced cerebral perfusion that occurs in heat-stressed individuals. *Significantly different relative to normothermia; §significantly different relative to control heat stress. Figure from Brothers et al. (2009b); republished with permission from Wiley-Blackwell.
Middle Cerebral Artery Occlusion Models, supplied by Van Hoef Consulting, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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middle cerebral artery occlusion models - by Bioz Stars, 2026-08
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CEM Corporation middle cerebral artery occlusion sites
End-tidal carbon dioxide tension and <t>middle</t> <t>cerebral</t> <t>artery</t> <t>blood</t> <t>velocity</t> (MCA Vmean) during normothermia, heat stress, and heat stress after end-tidal carbon dioxide (PETCO2) concentration was returned to pre-heat stress levels. The reduction in PETCO2 concentration during heat stress was completely abolished by the PETCO2 clamping procedure (panel a). Heat stress reduced MCA Vmean relative to normothermia. Restoration of PETCO2 to the normothermic level while subjects were heat stressed (heat stress + clamp) attenuated the decrease in MCA Vmean relative to control heat stress without the clamp; however MCA Vmean remained reduced when compared with normothermia (panel b). These data indicate that mechanisms other than reduced PETCO2 contribute to the reduced cerebral perfusion that occurs in heat-stressed individuals. *Significantly different relative to normothermia; §significantly different relative to control heat stress. Figure from Brothers et al. (2009b); republished with permission from Wiley-Blackwell.
Middle Cerebral Artery Occlusion Sites, supplied by CEM Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/middle+cerebral+artery+infarction/pmc06493577-25-27-36?v=CEM+Corporation
Average 90 stars, based on 1 article reviews
middle cerebral artery occlusion sites - by Bioz Stars, 2026-08
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Segeberger Kliniken middle cerebral artery (mca)
End-tidal carbon dioxide tension and <t>middle</t> <t>cerebral</t> <t>artery</t> <t>blood</t> <t>velocity</t> (MCA Vmean) during normothermia, heat stress, and heat stress after end-tidal carbon dioxide (PETCO2) concentration was returned to pre-heat stress levels. The reduction in PETCO2 concentration during heat stress was completely abolished by the PETCO2 clamping procedure (panel a). Heat stress reduced MCA Vmean relative to normothermia. Restoration of PETCO2 to the normothermic level while subjects were heat stressed (heat stress + clamp) attenuated the decrease in MCA Vmean relative to control heat stress without the clamp; however MCA Vmean remained reduced when compared with normothermia (panel b). These data indicate that mechanisms other than reduced PETCO2 contribute to the reduced cerebral perfusion that occurs in heat-stressed individuals. *Significantly different relative to normothermia; §significantly different relative to control heat stress. Figure from Brothers et al. (2009b); republished with permission from Wiley-Blackwell.
Middle Cerebral Artery (Mca), supplied by Segeberger Kliniken, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Subgroup analyses (functional outcome)

Journal: The Cochrane Database of Systematic Reviews

Article Title: Endovascular thrombectomy and intra‐arterial interventions for acute ischaemic stroke

doi: 10.1002/14651858.CD007574.pub3

Figure Lengend Snippet: Subgroup analyses (functional outcome)

Article Snippet: A total of 20 participants were lost to follow‐up across all 19 studies. . Types and severities of strokes Three studies included participants with middle cerebral artery territory strokes ( MELT 2007 ; PROACT 1 1998 ; PROACT 2 1999 ).

Techniques: Functional Assay

MELT 2007

Journal: The Cochrane Database of Systematic Reviews

Article Title: Endovascular thrombectomy and intra‐arterial interventions for acute ischaemic stroke

doi: 10.1002/14651858.CD007574.pub3

Figure Lengend Snippet: MELT 2007

Article Snippet: A total of 20 participants were lost to follow‐up across all 19 studies. . Types and severities of strokes Three studies included participants with middle cerebral artery territory strokes ( MELT 2007 ; PROACT 1 1998 ; PROACT 2 1999 ).

Techniques: Disruption, Comparison, Sequencing, Selection, Modification

MR CLEAN 2015

Journal: The Cochrane Database of Systematic Reviews

Article Title: Endovascular thrombectomy and intra‐arterial interventions for acute ischaemic stroke

doi: 10.1002/14651858.CD007574.pub3

Figure Lengend Snippet: MR CLEAN 2015

Article Snippet: A total of 20 participants were lost to follow‐up across all 19 studies. . Types and severities of strokes Three studies included participants with middle cerebral artery territory strokes ( MELT 2007 ; PROACT 1 1998 ; PROACT 2 1999 ).

Techniques: Biomarker Discovery, Disruption, Modification, Sequencing, Selection

PROACT 1 1998

Journal: The Cochrane Database of Systematic Reviews

Article Title: Endovascular thrombectomy and intra‐arterial interventions for acute ischaemic stroke

doi: 10.1002/14651858.CD007574.pub3

Figure Lengend Snippet: PROACT 1 1998

Article Snippet: A total of 20 participants were lost to follow‐up across all 19 studies. . Types and severities of strokes Three studies included participants with middle cerebral artery territory strokes ( MELT 2007 ; PROACT 1 1998 ; PROACT 2 1999 ).

Techniques: Isolation, Imaging, Sequencing, Selection, Control, Saline

NCT02419781

Journal: The Cochrane Database of Systematic Reviews

Article Title: Endovascular thrombectomy and intra‐arterial interventions for acute ischaemic stroke

doi: 10.1002/14651858.CD007574.pub3

Figure Lengend Snippet: NCT02419781

Article Snippet: A total of 20 participants were lost to follow‐up across all 19 studies. . Types and severities of strokes Three studies included participants with middle cerebral artery territory strokes ( MELT 2007 ; PROACT 1 1998 ; PROACT 2 1999 ).

Techniques: Modification

A 61-year old female presented with a right parietal hemorrhage, generalized tonic clonic seizure, and left hemiparesis. She was found to harbor an AVM at the site of hemorrhage and was treated with GKR 1 month after presentation. Several diagnostic cerebral angiogram images are presented. A.) Anterio-posterior (AP) projection of an internal carotid artery (ICA) injection. Poor detailed arterial, nidal, and venous resolution of the AVM are noted (White arrow). B.) Anterio-posterior (AP) projection of a selective middle cerebral artery (MCA) injection. Poor detailed arterial, nidal, and venous resolution of the AVM are noted (White arrow). C.) 3DRA right ICA injection with a right parietal AVM (white arrow) but with continued limited arterial, nidal, and venous anatomy. D.) Axial CBCT-A reconstruction with patient in the Leksell head frame. The anterior posts and posterior pins of the frame are seen. A right parietal AVM is visualized. E.) Magnified axial CBCT-A reconstruction. Excellent resolution of the complex arterial and venous structure of the AVM are noted (White arrow). F.) Screen shot image of a coronal CBCT-A reconstruction uploaded for planning on the Leksell stereotactic planning computer. The radiated field is demonstrated by the encircled areas at different radiation isodoses. Note that the draining vein is easily resolved and left out of the radiation field (White arrow). G.) 2-year follow-up AP projection of an ICA injection demonstrating complete obliteration of the previously seen AVM. H.) 2-year follow-up magnified axial CBCT-A reconstruction demonstrating complete obliteration of the previously seen AVM.

Journal: Neurosurgery

Article Title: Utilization of Cone-Beam Computed Tomographic Angiography in Planning for Gamma Knife Radiosurgery of Arteriovenous Malformations: A Case Series and Early Report

doi: 10.1227/NEU.0000000000000331

Figure Lengend Snippet: A 61-year old female presented with a right parietal hemorrhage, generalized tonic clonic seizure, and left hemiparesis. She was found to harbor an AVM at the site of hemorrhage and was treated with GKR 1 month after presentation. Several diagnostic cerebral angiogram images are presented. A.) Anterio-posterior (AP) projection of an internal carotid artery (ICA) injection. Poor detailed arterial, nidal, and venous resolution of the AVM are noted (White arrow). B.) Anterio-posterior (AP) projection of a selective middle cerebral artery (MCA) injection. Poor detailed arterial, nidal, and venous resolution of the AVM are noted (White arrow). C.) 3DRA right ICA injection with a right parietal AVM (white arrow) but with continued limited arterial, nidal, and venous anatomy. D.) Axial CBCT-A reconstruction with patient in the Leksell head frame. The anterior posts and posterior pins of the frame are seen. A right parietal AVM is visualized. E.) Magnified axial CBCT-A reconstruction. Excellent resolution of the complex arterial and venous structure of the AVM are noted (White arrow). F.) Screen shot image of a coronal CBCT-A reconstruction uploaded for planning on the Leksell stereotactic planning computer. The radiated field is demonstrated by the encircled areas at different radiation isodoses. Note that the draining vein is easily resolved and left out of the radiation field (White arrow). G.) 2-year follow-up AP projection of an ICA injection demonstrating complete obliteration of the previously seen AVM. H.) 2-year follow-up magnified axial CBCT-A reconstruction demonstrating complete obliteration of the previously seen AVM.

Article Snippet: Anterio-posterior (AP) projection of a selective middle cerebral artery (MCA) injection.

Techniques: Diagnostic Assay, Injection

End-tidal carbon dioxide tension and middle cerebral artery blood velocity (MCA Vmean) during normothermia, heat stress, and heat stress after end-tidal carbon dioxide (PETCO2) concentration was returned to pre-heat stress levels. The reduction in PETCO2 concentration during heat stress was completely abolished by the PETCO2 clamping procedure (panel a). Heat stress reduced MCA Vmean relative to normothermia. Restoration of PETCO2 to the normothermic level while subjects were heat stressed (heat stress + clamp) attenuated the decrease in MCA Vmean relative to control heat stress without the clamp; however MCA Vmean remained reduced when compared with normothermia (panel b). These data indicate that mechanisms other than reduced PETCO2 contribute to the reduced cerebral perfusion that occurs in heat-stressed individuals. *Significantly different relative to normothermia; §significantly different relative to control heat stress. Figure from Brothers et al. (2009b); republished with permission from Wiley-Blackwell.

Journal: Acta physiologica (Oxford, England)

Article Title: Cardiovascular function in the heat-stressed human

doi: 10.1111/j.1748-1716.2010.02119.x

Figure Lengend Snippet: End-tidal carbon dioxide tension and middle cerebral artery blood velocity (MCA Vmean) during normothermia, heat stress, and heat stress after end-tidal carbon dioxide (PETCO2) concentration was returned to pre-heat stress levels. The reduction in PETCO2 concentration during heat stress was completely abolished by the PETCO2 clamping procedure (panel a). Heat stress reduced MCA Vmean relative to normothermia. Restoration of PETCO2 to the normothermic level while subjects were heat stressed (heat stress + clamp) attenuated the decrease in MCA Vmean relative to control heat stress without the clamp; however MCA Vmean remained reduced when compared with normothermia (panel b). These data indicate that mechanisms other than reduced PETCO2 contribute to the reduced cerebral perfusion that occurs in heat-stressed individuals. *Significantly different relative to normothermia; §significantly different relative to control heat stress. Figure from Brothers et al. (2009b); republished with permission from Wiley-Blackwell.

Article Snippet: Therefore, it is plausible to speculate that heat stress-induced increases in cerebral sympathetic activity may contribute to reductions in cerebral perfusion in the heat-stressed human. fig ft0 fig mode=article f1 fig/graphic|fig/alternatives/graphic mode="anchored" m1 Open in a separate window Figure 5 caption a7 End-tidal carbon dioxide tension and middle cerebral artery blood velocity (MCA V mean ) during normothermia, heat stress, and heat stress after end-tidal carbon dioxide (PETCO 2 ) concentration was returned to pre-heat stress levels.

Techniques: Concentration Assay, Control

Cerebral circulation and oxygenation during maximal whole-body exercise in heat-stressed humans. Left and right middle cerebral artery blood velocity and near-infrared spectroscopy-determined cerebral tissue oxygenation at rest, during submaximal and maximal cycling and during 10 min of recovery in heat stress and control conditions. Note the marked reductions in blood velocity accompanying the declines in tissue oxygenation. *Higher than value at start of exercise, P < 0.05. †Lower than peak value during maximal exercise, P < 0.05. From González-Alonso et al. (2004); republished with permission from Wiley-Blackwell.

Journal: Acta physiologica (Oxford, England)

Article Title: Cardiovascular function in the heat-stressed human

doi: 10.1111/j.1748-1716.2010.02119.x

Figure Lengend Snippet: Cerebral circulation and oxygenation during maximal whole-body exercise in heat-stressed humans. Left and right middle cerebral artery blood velocity and near-infrared spectroscopy-determined cerebral tissue oxygenation at rest, during submaximal and maximal cycling and during 10 min of recovery in heat stress and control conditions. Note the marked reductions in blood velocity accompanying the declines in tissue oxygenation. *Higher than value at start of exercise, P < 0.05. †Lower than peak value during maximal exercise, P < 0.05. From González-Alonso et al. (2004); republished with permission from Wiley-Blackwell.

Article Snippet: Therefore, it is plausible to speculate that heat stress-induced increases in cerebral sympathetic activity may contribute to reductions in cerebral perfusion in the heat-stressed human. fig ft0 fig mode=article f1 fig/graphic|fig/alternatives/graphic mode="anchored" m1 Open in a separate window Figure 5 caption a7 End-tidal carbon dioxide tension and middle cerebral artery blood velocity (MCA V mean ) during normothermia, heat stress, and heat stress after end-tidal carbon dioxide (PETCO 2 ) concentration was returned to pre-heat stress levels.

Techniques: Spectroscopy, Control